›› 2003, Vol. 46 ›› Issue (6): 788-792.doi:
• RESEARCH PAPERS • Previous Articles Next Articles
ZHAO Wei-Dong, WANG Kai-Yun*, JIANG Xing-Yin, YI Mei-Qin
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Abstract: The selection for resistance by abamectin, pyridaben and fenpropathrin to Tetranychuc urticae were carried out in laboratory, as well as activity change of detoxicant enzymes. The susceptible population of T. urticae was separately treated with abamectin, pyridaben and fenpropathrin imitating field chemicals selection pressure in order to breed its resistance. The results showed that the resistance of T.urticae had reached 6.72fold to abamectin, 19.9fold to fenpropathrin and 12.1fold to pyridaben after 12 generations treatment. The resistance mechanism was evaluated by activity measurement of enzymic inhibitors and detoxicant enzymes. It was concluded that the rise of resistance to abamectin was mainly associated with increased activities of mixedfunction oxidases and glutathione S-transferase; the resistance to pyridaben was attributed to increased activIties of carboxylesterase and mixedfunction oxidases; the main cause of resistance to fenpropathrin was the increased activities of carboxylesterase and glutathione S-transferase.
Key words: Tetranychus urticae; abamectin, pyridaben, fenpropathrin, resistance selection, detoxicant enzymes
ZHAO Wei-Dong, WANG Kai-Yun*, JIANG Xing-Yin, YI Mei-Qin. Resistance selection by abamectin, pyridaben and fenpropathrin and activity change of detoxicant enzymes in Tetranychus urticae[J]., 2003, 46(6): 788-792.
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